Oxidative stress is caused by the unchecked activities of free radicals and reactive oxygen species (ROS). These are highly reactive, unstable molecules that, in healthy cells, are created and neutralized as part of normal cell life. However, if the production of free radicals and ROS exceeds the body's natural defense mechanisms, oxidative damage to vital molecules in the body can occur, leading to cell death.
OXIS' goal has been to control both direct cellular damage from ROS and the activation of certain molecules called transcription factors, which are activated by ROS and trigger inflammatory responses and cell death in degenerative diseases.
OXIS ( http://www.oxis.com ) prepares to bring a unique compound called Ergothioneine, one of the more powerful antioxidants available, into the highly lucrative nutraceuticals market. With its proprietary method for commercially producing Ergothioneine, OXIS plans to become the primary worldwide source for the molecule. Two product launches are planned for summer 2010: (1) Ergo-Pur - a pure form of our highly potent multifaceted antioxidant which we anticipate will be used in some cases in concert with, (2) Ergo-Plex - a combination of ERGO plus other functional nutraceutical ingredients directed at Joint Health.
Some of the potential benefits of ERGO include its ability to: (1) Conserve and maintain the levels of other antioxidants such as Vitamin E, Vitamin C and glutathione; (2) Increase respiration and the oxidation of fat (possibly contributing to increased energy and exercise capacity); (3) Protect mitochondria from damage; (4) Reduce the damaging effects of environmental ultraviolet radiation (likely to be important in protecting the eyes against cataract producing oxidative injury); (5) Neutralize increased oxidative stress by providing an ROS (reactive oxygen species) and RNS (reactive nitrogen species) scavenging capacity, a property that protects key molecules in the body; and, (6) Protect against the effects of neurotoxins that are believed to have a casual role in the development and progression of cognitive decline.
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